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Since the astrocytic K + uptake also depends on glycogenolysis-activated signaling (to facilitate Na + access to the Na + ,K + -ATPases Na + -sensitive site), it must also be absent in neonatal brain

After mucosal barrier disruption, DSS is phagocytosed by antigen-presenting cells (APCs) in the submucosal layer

The result is sustained, systemic IGF-1 receptor activation that bypasses the regulatory mechanisms controlling endogenous IGF-1 bioavailability

Apoptosis can be initiated via three principal pathways: the extrinsic (death receptor) pathway, the intrinsic (mitochondrial) pathway, and the endoplasmic reticulum (ER) stress pathway (Figure 2A)