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At the basis of this dysfunction, now termed adiposopathy, there would be a complex sequence of morphological and functional alterations including reduction of adipogenic potential with prevalent expansion due to hypertrophy, recruitment of inflammatory cells, alteration of the secretion pattern of adipocytokines, inflammation, hypoxia, fibrosis, and mitochondrial dysfunction [12, 13]
& Cuthbertson, D
Consumption of ultra-processed foods and body fat during childhood and adolescence: a systematic review
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