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The fact that this redox imbalance occurs as a consequence of GSH depletion may explain why several clinical trials have provided no evidence of significant cataract prevention arising from the administration of high dosages of ascorbic acid [13]

The majority of evidence regarding GLP-1 RAs therapy in patients with steroid-induced dysglycemia originates from cohort studies conducted in solid organ transplant recipients and oncological populations, with a limited number of case reports involving patients with rheumatic diseases

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