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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

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Osteosarcoma cell-derived migrasomes promote macrophage M2 polarization to aggravate osteosarcoma proliferation and metastasis

It is not surprising that AAS products constituted the largest group in each year between 2020 and 2024 (average: 68.4%)