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[9] By inhibiting somatostatin, DSIP may potentially contribute to hypertrophy and hyperplasia in skeletal muscle
11 Conversion to PDR was higher in the GLP-1 RA group at 1 and 3 years, along with high rates of DME at consistent time points up to 3 years
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Cardiomyocyte OTUD1 drives diabetic cardiomyopathy via directly deubiquitinating AMPK 2 and inducing mitochondrial dysfunction
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