Preterm infants may be particularly susceptible to OxS damage due to immaturity of the antioxidant defence systems, which are mainly represented by endogenous cellular antioxidant enzymes, e.g., catalase (CAT), glutathione reductase (GR), glutathione peroxidase (GPx), and superoxide dismutase (SOD), and exogenous/dietary non-enzymatic antioxidants (e.g., retinol, -carotene, and -tocopherol) (8, 9)
The staff are very supportive
Some patients use maintenance protocols for ongoing digestive support
Powder X-ray diffraction (PXRD) analyses of APTES-COF-1 revealed maintained crystallinity post-functionalization, with distinct (110), (201), and (002) diffraction planes confirming structural integrity during drug encapsulation [145]