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In animal models of heart failure induced by high-frequency pacing, ischaemia, and obesity, GLP-1RAs inhibit left ventricular remodelling and improve left ventricular contractility and diastolic function through anti-inflammatory effects, cardiomyocyte apoptosis inhibition, and glucose metabolism enhancement in cardiomyocytes [38,39,40]
TB-500 is studied primarily for its cellular migration-promoting, stem cell-mobilising, actin-sequestering, and anti-apoptotic properties across wound healing, cardiac, CNS, and musculoskeletal models
Mitrea, L., Neme, S.-A., Szabo, K., Teleky, B.-E
Y.WangL.-F.WuC.-H.KeC.-H.et al (2014)
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