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In preclinical studies on tissues like tendons and muscles, significant improvements in healing and function are often observed within a few weeks
This mechanism involves: Control of actin filament assembly and disassembly Enhanced cellular migration through cytoskeletal reorganization Improved cell morphological changes during repair processes Regulation of cellular mechanical properties The peptides actin-binding domain (LKKTET sequence) allows precise modulation of cytoskeletal dynamics, enabling cells to migrate more effectively to sites requiring repair
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