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The hydrolysis of extracellular Gln by the GGT from gastric helicobacters can cause a depletion of this conditionally essential amino acid in the gastric juice of an infected host, which may eventually lead to epithelial cell damage and immunological dysfunction 16,18,20
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It promotes lipolysis through growth hormone receptor pathways, which differs from the NNMT inhibition mechanism of 5-amino-1mq
Int J Mol Sci 19(7):1987
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