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Copper-mediated neurotoxicity and genetic vulnerability in the background of neurodegenerative diseases in C
The 2013 Journal of Neurochemistry paper by McCoy et al
Caloric restriction and the aging process: a critique
Here, we demonstrated consistent metabolic benefits of an oral, gut- and liver-restricted LXR inverse agonist, TLC-2716, in rodent, NHP and HLO models and, ultimately, in healthy human participants, supporting the potential of this approach for the treatment of cardiometabolic diseases
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