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All three conditions, obesity without eating disorders, obesity with BED, and BN, are characterized by disruptions in appetite regulation, reward processing, and metabolic homeostasis
They work by mimicking a natural hormone that helps regulate appetite and promotes a feeling of fullness, making it easier to reduce calorie intake
The results indicated that GSTM2 regulated SREBF1 and PLIN2 expression by affecting ASK1 activity
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