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Mechanistically, PYC inhibits NF-B activation, leading to decreased transcription of pro-inflammatory mediators such as TNF-, ICAM-1, and vascular cell adhesion molecule-1, thereby modulating cytokine-driven inflammatory responses and endothelial activation ( Network pharmacology and protein-protein interaction analyses have identified IL-6, TNF, IL-1B, prostaglandin endoperoxide synthase 2, and NF-B1 as core molecular targets of PYC, with Kyoto Encyclopedia of Genes and Genomes (KEGG) enrichment highlighting the IL-17 and TNF signaling pathways as key anti-inflammatory mechanisms
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This data can be found here: The University of Aberdeen Research Portal, DOI: 10.20392/aef8fe2e-5180-4693-b187-750dfecf5e9e
Interactions With Other Therapies and Medical Conditions Peptides may interact with HRT plans, Thyroid Management strategies, or medications affecting growth hormones
