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Last reviewed: June 3, 2026 Last updated: June 3, 2026 Written by: Jay Hastings, CEO of PlexusDx Jay Hastings is the CEO of PlexusDx, a precision health company focused on genetic testing, blood biomarker insights, and personalized wellness recommendations
While genetic syndromes characterized by leptin deficiency present hyperfagia and obesity (Zhang et al., 1994), most obese individuals rather have hyperleptinemia (Schwartz et al., 1997), due to desensitization of its own receptor (Considine et al., SNS is involved in regulation of secretory function of WAT, especially for leptin secretion
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[5] Beyond its antioxidant properties, ALA may serve as an essential cofactor for mitochondrial enzymatic complexes (notably the -ketoacid dehydrogenases) involved in oxidative metabolism
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