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Hypothalamic-pituitary-adrenal hypofunction in myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS) as a consequence of activated immune-inflammatory and oxidative and nitrosative pathways
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Although the exact mechanism of action is not fully understood, it is hypothesised that bupropion stimulates hypothalamic pro-opiomelanocortin (POMC) cell, with downstream effects to reduce food intake and increase energy expenditure
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Adipose expression of tumor necrosis factor-: direct role in obesity-linked insulin resistance
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