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Deletion of gsh-px gene increased concentrations of GSH in the mutant, which improved the efficiency of allosteric binding of PrfA to GSH and enhanced the regulation of virulence proteins like InlA, InlB and LLO (Kim et al., 2005), increasing pathogenicity of the gsh-px deletion strain
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Copper peptides contribute growth factor signaling, wound healing support, broad gene expression modulation, and anti-inflammatory action
showed that maternal hyperglycemia reduces the expression of IGF-1, resulting in delayed fetal dendrite development in STZ-induced rats [125]
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