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Therefore, Alzheimers disease (AD), characterized by cognitive impairment and memory loss, is inevitably influenced by the accumulation of TMAO
At present, several GLP-1 analogs have joined the class, such as exenatide, liraglutide, dulaglutide, semaglutide and tirzepatide [1]
The glucagon-like peptide-1 receptor agonist reduces inflammation and bloodbrain barrier breakdown in an astrocyte-dependent manner in experimental stroke
The first GLP-1 receptor agonist, exenatide, was developed in 2005 and safely prescribed ever since
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