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Mitochondrial dysfunction and oxidative stress in Alzheimers disease: Implications for mitochondria-targeted antioxidant therapeutics

this pathway has been shown to impair process outgrowth in neurons and regulate glycolysis - the major source of of ATP in mature OLs which contributes to protein and lipid production needed for myelin biosynthesis

548 Under conditions of normal oxygen levels (normoxia), the activity of HIF-1 is suppressed via redox-dependent modifications

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