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It also plays a crucial role in the production of endothelium-derived relaxing factor (EDRF) , primarily nitric oxide

Ototoxic agents impair mitochondrial function, disrupt ATP synthesis, and activate caspase-mediated apoptosis via the intrinsic (mitochondrial) pathway, leading to irreversible auditory cell loss

The insulin response from eating antagonizes the fat-releasing pathways the peptide activates

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