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The widespread distribution and function of multiple GLP-1 receptor populations in the central and autonomic nervous system provide the anatomical foundation for the appetite-suppressing effects observed with GLP-1 agonist therapy
359,360 Functional abnormalities also play a critical role in DPN, including altered thalamic activity and disrupted thalamocortical connectivity
Strong food noise, cravings, or appetite dysregulation that has not responded well to nutrition changes alone
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