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Glucagon-like peptide-1 causes pancreatic duodenal homeobox-1 protein translocation from the cytoplasm to the nucleus of pancreatic beta-cells by a cyclic adenosine monophosphate/protein kinase A-dependent mechanism
However, NO is not universally detrimental
Children on these medications need careful monitoring to ensure adequate protein for growth, sufficient calories for development, proper micronutrient intake, and maintenance of bone density
Residual beta-cell function more than glycemic control determines abnormalities of the insulin-like growth factor system in type 1 diabetes
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