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(4) Schizophrenia (associated with reduced HGF/c-Met signaling in postmortem brain studies)

HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use

Protein synthesis Testosterone can also have beneficial effects in increasing the production of protein in muscles

Gillessen, A., & Schmidt, H