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Combination Therapies: PLE enhances outcomes when paired with other treatments
Selective impairment of this carrier-mediated transport can contribute to selective vulnerability of tissues to specific toxins: for example, chronically ethanol-fed hepatocytes become selectively depleted of mitochondrial glutathione because of defective carrier-mediated transport, which makes the hepatocytes susceptible to damage by reactive oxygen species generated from the oxidative metabolism of ethanol (22)
It reverses the cell damage caused by fatty liver disease Cell death in the liver is aggravated by a deficiency in antioxidants, including glutathione
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