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As expected, silencing of endogenous KEAP1 increased luciferase activity driven by ARE elements under Cyss depletion conditions, whereas simultaneous expression of exogenous shRNA-resistant KEAP1 restored basal expression (Fig
Moreover, its ability to modulate serotonin and dopamine pathways suggests potential antidepressant properties, offering hope for individuals grappling with mood disorders stemming from neurological conditions
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