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There are around 80 different autoimmune disorders ranging in severity from mild to disabling, depending on which system of the body is under attack and to what degree
ROS-induced NO inactivation can impair vascular endothelial function, contributing to various pathologies like atherosclerosis, hypertension, cardiovascular disease, and central nervous system disorders (27, 28)
This approach is pragmatic when blocks are missing or modalities differ substantially in measurement properties, but it should not be taken as mechanistic evidence without transparent attribution within each modality and explicit evaluation of calibration and transportability across centres [459,460,461]
Adjusting protocols based on progress requires thoughtful interpretation of available data
