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79 Rather than depending on a single relay mechanism, extracellular ferroptotic propagation is likely mediated by a context-dependent signaling network whose dominant components vary with tissue architecture and disease state

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Epithelial cells prime the immune response to an array of gut-derived commensals towards a tolerogenic phenotype through distinct actions of thymic stromal lymphopoietin and transforming growth factor-

Hepcidin can directly bind to the membrane-bound FPN, promoting its internalization and degradation in intestinal epithelial cells, thus inhibiting iron absorption into the blood