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doi: 10.1111/imj.16388 44 LiYTanYXiaGShuaiJ
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Compared to BPC-157, which acts primarily via the nitric oxide and growth hormone receptor pathways, and TB-500, which functions through actin sequestration and VEGF signalling, GHK-Cu operates largely through copper-mediated gene expression modulation and extracellular matrix remodelling
This is particularly true among sulfur-containing residues such as methionine and cysteine
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