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Amyloid plaque is primarily formed of Abeta fibrils, and much research on potential therapies has centered on creating anti-amyloid monoclonal antibodies, with the aim of clearing the plaque by targeting the peptide for degradation ( A fascinating paper by Serra-Vidal et al
Oral administration must be carefully managed to avoid gastrointestinal adverse reactions
Researchers found that 37 percent of people on the drug saw liver scarring improve, compared with about 23 percent on the placebo
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