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Key properties: Stimulates GH release from the pituitary (not the hypothalamus) Does not significantly raise cortisol or prolactin Short half-life (~2 hours) produces a sharp, pulsatile GH release Does not block somatostatin (the hormone that inhibits GH release) Selective for GH minimal effect on other hormones The selectivity is what makes ipamorelin the preferred GHRP for most protocols

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The combination addresses multiple aspects of a metabolic-neurological clinical picture without competing for receptor or pathway

When administered through injection, GLP-1 medications stimulate the same receptors in the brain and gut that signal fullness, slow gastric emptying, and reduce the desire to overeat