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Increasing availability of -3 fatty acid in the early-life diet prevents the early-life stress-induced cognitive impairments without affecting metabolic alterations

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Thus, damage to mitochondrial DNA and loss of can further increase ROS production, thereby creating a cycle of dysfunction that includes increased ROS production by the respiratory chain, activation of signal transduction pathways, and potentially, leading to apoptosis or necrosis, as well as the release of cytochrome c, which triggers apoptosis