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However, while our results corroborate the existence of GLP-1R-associated endosomal signalling, they also suggest that sustained GLP-1R signalling, as indicated by cumulative insulin secretion over pharmacologically relevant timescales, is actually greater with the ligand (exendin-4-C16) with a reduced tendency to recruit mini-G s to Rab5-positive endosomes

Perhaps they are not frequent enough.Do you have other symptoms Some symptoms overlap with many other things

But the same location in the peroxidase enzymes is blocked by the protein structure so that substrates can interact only with the heme edge