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Furthermore, Gclc -deficient FoB have a block in the mitochondrial electron transport chain (ETC) due to diminished complex I and II activity and thereby accumulate the tricarboxylic acid cycle metabolite succinate

I like it best, the way Lenzi A et al said it in their paper Rationale for Glutathione Therapy published in the Oxford journal Human Reproduction

Recent studies also highlight the potential of leveraging the bonebrain axis as a novel route for SIN delivery, offering enhanced targeting of ischemic brain tissue

This designation indicates the FDA recognizes its potential for treating conditions that currently lack adequate treatments