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That mechanism is plausible, but a copper complex acting in cell models does not automatically prove the same outcome in human patients
Its a question that seems simple on the surface, yet its one that lands in our inbox with surprising frequency
A variation in t max was observed across the groups but with no apparent association with increasing hepatic impairment (45, 36, 84 and 45 h for normal hepatic function and mild, moderate and severe hepatic impairment, respectively)
Similarly, IL-36, a cytokine that can induce fibrosis, is found at higher levels in fibrotic intestinal tissues of CD patients (160)
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