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Through the employment of adenoviral constructs for NR4a1 knockout and overexpression, we validated Semas beneficial effects on glucolipid metabolism and mitochondrial dysfunction in pathological myocardial remodeling via NR4a1 modulation (Figs
Treatment discontinuation among users of GLP-1 receptor agonists and SGLT2 inhibitors in a national population of individuals with type 2 diabetes
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