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In 3 of the studies, patients were treated with liraglutide, whereas exenatide was used in the other 2 studies
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This suggests that, contrary to what has been suggested by Freeman et al [26] but in agreement with the seminal paper by Toye et al in which the impact of NNT truncation with seven other genetic loci only accounted for 35% of the increase in IPGTT AUC in BL/6J mice [15], NNT is responsible for only a small part (~1520%) of the increase in IPGTT AUC in BL/6J vs BL/6N female mice, a proportion that may rise to about 30% in males
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