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A landmark study published in Diabetes (Tolhurst et al., 2012) demonstrated that SCFAs stimulate GLP-1 secretion via the FFAR2 receptor, and that mice lacking this receptor showed significantly reduced SCFA-triggered GLP-1 release
And that connection is very real
When to Contact Your Healthcare Provider Most people can complete a course of antibiotics without any issues while remaining on their GLP-1 program
A separate set of enzymes and transporters also plays a buffering role by eliminating acetyl-CoA from inside mitochondria created by the pyruvate dehydrogenase complex that is in excess of its utilization by the Krebs cycle
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