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In isolated cardiomyocytes, GLP-1 receptor agonists activated PI3K/Akt, AMPK, and cAMP/PKA pathways while reducing NOX4-mediated oxidative stress, signalling cardioprotective effects by alleviating ER stress-induced apoptosis [51]

In a recent study, 54 patients with knee OA were randomly assigned to three treatment groups and their knees were injected with either PRP, Hyaluronic Acid, or a peptide solution