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Mitochondrial dysfunction and oxidative stress are often implicated in progressive neurodegenerative disorders such Alzheimers disease and Parkinsons disease and in damage secondary to TBI.5,6 Additionally, new research both in vitro and in mice has suggests that methylene blue may reduce neuronal apoptosis and improve blood-brain barrier integrity.7 One randomized, blinded, placebo-controlled trial evaluated the effects of methylene blue 10mg/kg administered intraperitonially to mice and rats immediately or 24 hours post ischemic stroke

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provide new insights into the regulatory role of SUV39H1 in the progression of cancer, and show that SUV39H1 inhibits the transcription of dipeptidyl-peptidase-4 (DPP4) through catalyzing the methylation of DPP4 promoter, therefore reducing the production of lipid ROS and repressing ferroptosis [84, 85]

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