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In LPS-treated BV2 cells and rat primary microglia, UA inhibited the production of pro-inflammatory cytokines (e.g., NO, TNF-, and PGE2) to shield dopaminergic neurons from the toxic effects of activated microglia

doi: 10.3389/fendo.2023.1215755 87 LiJChenQWangJHuangGYeH

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