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Alternative repletion strategies have focused on oral administration of GSH precursors (e.g., cysteine and glycine supplementation 9 ), and intravenous administration of GSH, 10 which although promising, is invasive and inconvenient, and therefore unlikely to be a practical solution

Huang et al., 2015), much like it is effective also in counteracting bleeding disorders (Stupnisek et al., 2012

An increase in the nuclear translocation of CaMKIV/CREM decreases GPX4, which is accompanied by an increase in intracellular lipid-ROS, eventually leading to neutrophil iron apoptosis (170) ( Figure 3 )

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