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To keep the bacterial count low, the small intestine employs its motility to sweep bacteria along, mucus, and antibacterial molecules secreted in the gastric acid, biliary juice, as well as substances produced by the commensal microflora and epithelial cells, such as Paneth cells. source Summary: The small intestine has built in defenses to keep bacterial overgrowth at bay

For instance, in Alzheimers disease (AD) models, sulforaphane administration enhanced antioxidant defense, reduced oxidative damage and improved behavioral outcomes

Reduced SIRT1 activity correlates with mtDNA damage, mitochondrial fragmentation, decreased membrane potential, and impaired adenosine triphosphate synthesis, all contributing to disease progression and transition to MASH

Footnotes This article has been peer reviewed