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By exploiting the self-assembly characteristics and anti-tumor activity of these compounds, they managed to significantly extend the plasma half-life of PTX and ursolic acid, thereby preventing fast drug leakage within the body

Structural modifications, such as attaching a lipid chain to a lysine residue, can enhance the peptides effectiveness by prolonging its half-life and bioavailability through non-covalent interactions with albumin

Fatty liver / MASH phenotype glucagon agonism plus GLP-1 agonism (survodutide, mazdutide, pemvidutide)