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These implications have been described across endocrine signaling pathways, receptor localization patterns, and intracellular cascade activation profiles, without implying translational relevance

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Myotubes could be induced to hypertrophy by the PI3K/Akt pathway, which increases protein synthesis and blocks the up-regulation of MAFbx and MuRF1 that occurs during muscle atrophy [54, 59]

This peptide inhibited TGF-1/Smad2/3 and STAT3 pathways, leading to decreased inflammation, tubular atrophy, myofibroblast activation, and fibrotic gene expression (Gwon et al., 2021)