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The biological effects attributed to this peptide appear to arise from its interaction with several interconnected physiological pathways

Experimental work in isolated vascular tissue and cell culture models indicates interactions involving regulators such as endothelial nitric oxide synthase, Src kinase signaling, and vascular endothelial growth factor receptor pathways

This effect is dose-dependent and fructose malabsorption is no longer detectable at a 1:1 stoichiometry in the fructose to glucose ratio (Reference Truswell, Seach and Thorburn10)

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