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The data conclusively demonstrated that both ME/CFS and Long COVID are characterized by severe, elevated oxidative stress and profound mitochondrial damage, forcing immune cells to consume excess host energy
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Site-directed mutagenesis experiments demonstrated the direct binding of NFAT at five sites of the GHRH promoter, suggesting that NFAT is involved in the depolarization-induced (with high potassium) transcriptional activation of GHRH gene in the hypothalamic neurons producing it [17]
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