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GPX7 can utilize the H 2 O 2 produced by Ero1 to accelerate the oxidative folding process of proteins in vitro and in vivo (Wang et al., 2014) (Figure 6)
The new tissue shows better organization and cross-linking, potentially creating stronger repairs than would occur naturally
Recent studies have identified specific Exos-mediated pathways critical for ferroptosis, offering a novel strategy for its clinical applications [17, 231]
Tryptophan deficiency arrests chromatin breakdown in secondary lens fibers of rats
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