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Omics-based studies have increasingly identified convergent endometrial and systemic pathways, including defective decidualization, immune dysregulation, abnormal extracellular matrix remodeling, and perturbed inflammatory signaling, that distinguish these conditions from isolated implantation or pregnancy failure and motivate a shift from single-marker interpretations toward multi-layer endotyping

Chronic Stress: High and prolonged stress levels can produce an excess of cortisol and other stress hormones that create oxidative byproducts

and they exacerbate renal injury and accelerate NS progression

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