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A particularly informative pharmacological example is provided by the diazepine derivative YL3147, a highly potent radical-trapping antioxidant that directly interrupts the chain-propagation phase of lipid peroxidation and confers robust protection in both acute and chronic murine models of doxorubicin-induced cardiomyopathy

In vitro experiments have shown that GSH deficiency induced by either the siRNA approach or pharmacological inhibition of the GCL rate-limiting enzyme decreases GSH biosynthesis and escalates oxidative stress in cultured hepatocytes

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This mesotherapy approach requires medical training but can produce dramatic localized results