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Uric acid treatment after stroke modulates the Krppel-like factor 2-VEGF-A axis to protect brain endothelial cell functions: impact of hypertension

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Notably, histological analysis at this time point revealed no significant hepatocyte damage, suggesting that the rapid decrease in hepsin protein levels may primarily result from the activation and subsequent degradation of the hepsin serine protease in response to APAP-induced hepatotoxicity

Clinical Pharmacokinetics of Semaglutide: A Systematic Review. Drug Design, Development and Therapy , vol