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Akt undergoes phosphorylation at Ser473 and Thr308, resulting in its active form, p-Akt, which subsequently phosphorylates FoxO1
However, the nonenzymatic formation of roGFP(S 2 ) from roGFP2(SSG) also proceeds extremely rapidly and competes with the final enzyme-catalyzed reaction step with implications for redox measurements as outlined below
Moreover, the majority of these early non-responders (53-70%) fail to achieve clinically meaningful weight loss of 5% after six months of treatment
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