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Through its interaction with NOTCH1, CHAC1/BOTCH inhibits NOTCH1 activity and thereby prevents neuronal stem cell self-renewal ( CHAC1/BOTCH through the transcription factor NRF2 and ROS signaling, which in turn inhibits stem cell self-renewal and promotes cell differentiation ( 2.4.1 The antagonistic roles of CHAC1 and CHAC2 in stem cell regulation The function of CHAC1 is further refined by its competitive interaction with its homolog, CHAC2
Mol Immunol 25(12):13471354 Carrington DM, Earl HS, Sullivan TJ (1987) Studies of human IgE to a sulfonamide determinant
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